Qualifications
MA (Cantab), FRCP, PhD
About Dr Morganstein
Consultant Endocrinologist at Chelsea and Westminster and The Royal Marsden hospitals and Honorary Clinical Senior Lecturer at Imperial College, with a special interest in endocrine oncology.
Dr Daniel Morganstein studied at Cambridge and qualified from St Bartholomew's and the Royal London in 1998. He trained in endocrinology and diabetes on the Hammersmith Rotation and undertook research at Imperial College London, where he attained his PhD.
He is experienced in all aspects of diabetes and endocrinology. His particular interests include endocrine tumours such as neuroendocrine tumours, adrenal tumours and paragangliomas; thyroid cancer, including medullary thyroid cancer; and the management of diabetes and hormonal effects during cancer treatment.
His further clinical interests include type 2 diabetes, pituitary and parathyroid disease. Dr Morganstein also provides diabetes and endocrinology services to several institutions and has published numerous scientific papers.
Dr Morganstein has been an invited speaker at a number of national and international scientific meetings including the British Endocrine Society, the European Endocrine Society, the European Association for Study of Diabetes and Diabetes UK Professional Conference.
MA (Cantab), FRCP, PhD
General Medical Council · 4518925
Consulting locations
Dr Morganstein provides services at the following hospitals.
Private Patients’ Department
369 Fulham Road, London, SW10 9NH
Hospital website View map280 King’s Road, London, SW3 5AW
Hospital website View map203 Fulham Road, London, SW3 6JJ
Hospital website View map19a Cavendish Square, London, W1G 0PL
Hospital website View mapPart of HCA Healthcare UK
Chelsea Bridge Road, London, SW1W 8RH
Hospital website View mapDr Morganstein's work spans endocrine oncology, diabetes in cancer care, thyroid disease and the management of hormone-related complications of cancer treatment.
.“Evaluating the Effectiveness of the Reducing Weight With Intensive Dietary Support (REWIND) Programme on Type 2 Diabetes Remission”.Diabetes Obes Metab.
.“To screen or not to screen: complexity of SDHA mutation management”.Endocr Oncol, 6(1): e250048.
.“Acute severe hypocalcaemia after initiation of a selective RET-inhibitor in medullary thyroid cancer”.Endocr Oncol, 5(1): e240060.
.“Mortality Outcomes in People with Lung Cancer with and without Type 2 Diabetes: A Cohort Study in England”.Clinical Epidemiology, Volume 17: 681-692.
.“Using routinely collected patient data to study the impact of type 2 diabetes on breast cancer”.Endocr Oncol, 5(1): e240039.
.“Association between hyperglycaemia, diabetes complications and development of fibrotic conditions among people living with type 1 and type 2 diabetes in England: a retrospective cohort study using UK Clinical Resource Datalink Aurum and Hospital Episode Statistics”.BMJ Open, 15(10): e103426.
.“Pancreatic surgery and tertiary pancreatitis services warrant provision for support from a specialist diabetes team”.World J Diabetes, 15(4): 598-605.
.“Diagnostic criteria and proposed management of immune-related endocrinopathies following immune checkpoint inhibitor therapy for cancer”.Endocr Connect, 2023; 12: 2023/03/09. DOI: 10.1530/EC-22-0513.
.“Hyperglycaemia following immune checkpoint inhibitor therapy—Incidence, aetiology and assessment”.Diabetic Medicine, 2023. DOI: 10.1111/dme.15053.
.“Diabetes and cancer: Optimising glycaemic control”.J Hum Nutr Diet, 2023; 36: 504-513. 2022/06/25. DOI: 10.1111/jhn.13051.
.“UK recommendations for SDHA germline genetic testing and surveillance in clinical practice”.J Med Genet, 2023; 60: 107-111. 2022/03/10. DOI: 10.1136/jmedgenet-2021-108355.
.“Type 2 diabetes mellitus and efficacy outcomes from immune checkpoint blockade in patients with cancer”.Clinical Cancer Research, 2023. DOI: 10.1158/1078-0432.CCR-22-3116.
.“Metastatic Medullary Thyroid Cancer: The Role of 68Gallium-DOTA-Somatostatin Analogue PET/CT and Peptide Receptor Radionuclide Therapy”.J Clin Endocrinol Metab, 2021; 106: e4903-e4916. 2021/08/12. DOI: 10.1210/clinem/dgab588.
.“Endocrine toxicity of cancer immunotherapy: clinical challenges”.Endocr Connect, 2021; 10: R116-R124. 2021/02/06. DOI: 10.1530/EC-20-0489.
.“Endocrinopathies induced by immune checkpoint inhibitors: the need for clear endocrine diagnosis”.Lancet Oncol, 2021; 22: 905-907. 2021/07/02. DOI: 10.1016/S1470-2045(21)00305-3.
.“Pituitary enlargement following ipilimumab without long term endocrine dysfunction”.Curr Probl Cancer, 2021: 100710. 2021/02/25. DOI: 10.1016/j.currproblcancer.2021.100710.
.“Immune landscape, evolution, hypoxia-mediated viral mimicry pathways and therapeutic potential in molecular subtypes of pancreatic neuroendocrine tumours”.Gut, gutjnl-2020-2321.
.“Predicting development of ipilimumab-induced hypophysitis: utility of T4 and TSH index but not TSH”.J Endocrinol Invest, 2020/05/26.
.“Glucocorticoid use and complications following immune checkpoint inhibitor use in melanoma”.Clin Med (Lond), 2020; 20: 163-168.
.“Immunotherapy-induced endocrinopathies: assessment, management and monitoring”.Ther Adv Endocrinol Metab, 2019; 10: 2042018819896182.
.“SOCIETY FOR ENDOCRINOLOGY ENDOCRINE EMERGENCY GUIDANCE: Acute management of the endocrine complications of checkpoint inhibitor therapy”.Endocrine Connections, 7(7): G1-G7.
.“Type 2 Diabetes and Osteoporosis: A Guide to Optimal Management”.J Clin Endocrinol Metab, 102(10): 3621-3634.
.“Histologically Proven Myocardial Carcinoid Metastases: The Value of Multimodality Imaging”.Can J Cardiol, 33(10): 1336.e1339-1336.e1312.
.“Thyroid Abnormalities following the use of CTLA-4 and PD-1 Inhibitors in the treatment of melanoma”.Clin Endocrinol (Oxf), Epub ahead of publication.
.“Hyperglycemia and Phosphatidylinositol 3-Kinase/Protein Kinase B/Mammalian Target of Rapamycin (PI3K/AKT/mTOR) Inhibitors in Phase I Trials: Incidence, Predictive Factors, and Management”.Oncologist, 2016; 21: 855-860.
.“Resolution of severe hyponatraemia is associated with improved survival in patients with cancer”.BMC Cancer, 2015; 15: 163.
.“Pancreatic neuroendocrine tumors: a review”.Future Oncol (Lond), 2015; 11(5): 853-864.
.“Prevalence of diabetes in patients admitted to a cancer hospital”.The British Journal of Diabetes & Vascular Disease, 2012; 12(4): 178-180.
.“Human fetal mesenchymal stem cells differentiate into brown and white adipocytes: a role for ERRalpha in human UCP1 expression”.Cell Res, 2010; 20(4): 434-444.
.“The role of arterial stimulation and simultaneous venous sampling in addition to cross-sectional imaging for localisation of biochemically proven insulinoma”.Eur Radiol, 2009 Oct; 19(10): 2467-2473. PubMed PMID: 19597822.
.“A functional interaction between RIP140 and PGC-1alpha regulates the expression of the lipid droplet protein CIDEA”.Mol Cell Biol, 2008; 28(22): 6785-6795.
.“Conditionally immortalized white preadipocytes: a novel adipocyte model”.J Lipid Res, 2008; 49(3): 679-685.
.“Role of RIP140 in metabolic tissues: Connections to disease”.FEBS Letters, 2007; 582(1): 39-45.
.“Role of nuclear receptor coregulators in metabolism”.Expert Review of Endocrinology and Metabolism, 2007; 2: 797-807.
.“Receptor Interacting Protein 140 regulates expression of Uncoupling Protein 1 in adipocytes through specific Peroxisome Proliferator Activated Receptor isoforms and Estrogen-Related Receptor”.Mol Endocrinol, 2007; 21(7): 1581-1592.
.“Low TSH in a patient with primary hypothyroidism”.J R Soc Med, 2005; 98(9): 417-418.
.“Successful treatment of Type B insulin resistance in a patient with otherwise quiescent systemic lupus erythematosus”.Diabetic Medicine, 2005; 22(6): 814-815.
.“Isolated B-cell lymphoma of the pituitary region: a rare clinical entity”.Hosp Med, 2002; 63(5): 306-307.
.“Simultaneous relapse of Graves' disease and minimal change glomerular disease”.Nephrol Dial Transplant, 2002; 17(4): 666-668.
.“Suppression of Zinc Interstitial Ion Migration in ZnO Due To The Presence of Sodium Ions”.Electroceramics, ed. Lee, W. E. and Bell, A., pub. Institute of Materials.